Research use onlyFor laboratory and research purposes only — not for human consumption, medical, veterinary or diagnostic use.

PeptideStacks
Longevity & Anti-Aging

Epitalon + Humanin + MOTS-c Longevity Stack — Research Protocol

Three-peptide longevity research stack targeting telomerase activation, mitochondrial unfolded-protein response and metabolic ageing. Full dosing protocol and UK regulatory note.

3 peptides 4-week cycle intermediateUpdated 2026-05-16
Telomerase activationMitochondrial biogenesisPineal melatonin signallingMetabolic homeostasis

This is the canonical three-peptide longevity research stack — each compound targets a distinct, well-characterised mechanism of cellular ageing. The combination has emerged from independent research lines: Soviet-era pineal peptide work (Epitalon), Japanese mitochondrial-peptide discovery (Humanin), and Lee lab MOTS-c characterisation (UC Davis / USC).

Mechanism of action — each peptide

Epitalon (Epithalon) — mechanism of action

A synthetic tetrapeptide (Ala-Glu-Asp-Gly) modelled on epithalamin, a pineal-gland extract studied extensively in Soviet and Russian gerontology research. In animal models:

  • Activates telomerase in somatic cells, lengthening telomeres in serially-passaged human fibroblasts (Khavinson et al.).
  • Normalises circadian melatonin secretion in aged animals — the canonical Epitalon finding.
  • Modulates pineal gene expression related to neuroendocrine ageing.
  • Increases mean and maximum lifespan in repeated rodent cohort studies (10–25% extension reported).

Humanin — mechanism of action

A 24-amino-acid peptide encoded within the mitochondrial 16S rRNA gene. In published research:

  • Suppresses apoptosis induced by Aβ-amyloid (originally discovered in Alzheimer's pathology studies).
  • Activates STAT3 signalling via the FPRL1 / FPR3 receptor.
  • Reduces oxidative stress markers in cardiac and pancreatic tissue.
  • Declines with age — plasma levels correlate inversely with cellular senescence markers.

MOTS-c — mechanism of action

A 16-amino-acid mitochondrial-derived peptide encoded within mtDNA. In published animal-model research:

  • Activates AMPK — the master metabolic switch — independently of insulin or AICAR.
  • Improves glucose tolerance in high-fat-diet rodent models without changing food intake.
  • Translocates to the nucleus under metabolic stress, modulating nuclear gene expression.
  • Mimics exercise-induced metabolic adaptation at the gene-expression level.

Full research protocol — dosing table

PeptideDoseFrequencyTimingCycle length
Epitalon5–10 mgDaily SCEvening pre-melatonin10–20 days
Humanin5–10 mgEvery other day SCMorning4 weeks
MOTS-c5–10 mg3x weekly SCPre-fasted state4 weeks

Weekly research timeline

PeptideWk 1Wk 2Wk 3Wk 4
Epitalon10 mg10 mg10 mg (taper)
Humanin10 mg EOD10 mg EOD10 mg EOD10 mg EOD
MOTS-c10 mg 3x10 mg 3x10 mg 3x10 mg 3x

Routes: SC = subcutaneous · IM = intramuscular · IN = intranasal · Oral · Topical.

Safety profile & regulatory note

Where to source these research peptides

Each peptide in this stack has a dedicated research monograph on PeptideAuthority.co.uk and a research-grade SKU at PeptideBarn.co.uk. All compounds are sold strictly for in vitro research.

Frequently asked research questions

Each targets a distinct longevity hallmark: Epitalon (telomere attrition + circadian dysregulation), Humanin (mitochondrial unfolded protein response, MUPR), MOTS-c (mitochondrial-derived metabolic homeostasis). Together they cover both nuclear and mitochondrial axes of ageing biology in published animal models.

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